Dr. Banek is currently an Assistant Professor at the University of Arizona College of Medicine in the Department of Physiology. His laboratory is currently supported by an NIH R00 Award, primarily focused on delineating the roles of peripheral sympathetic and sensory nerves in mediating arterial pressure control and renal function under healthy and hypertensive conditions. He has received several awards from international and national scientific societies for his research, including the APS, most recently the Michael J. Brody Award and the New Investigator Award – both from the Neural Control and Autonomic Regulation Section of APS. In addition to running his Cardiovascular Neurophysiology research laboratory, Dr. Banek serves on several national society committees, including his current service as chair of the APS Trainee Advisory Committee. Dr. Banek earned his PhD in Human Physiology at the University of Oregon in 2014 where he focused on mitigating the development of preeclampsia through exercise or pharmacological mimetics of exercise under the mentorship of Drs. Christopher Minson and Jeffrey Gilbert. His post-doctoral training was in cardiovascular physiology under the mentorship of Dr. John Osborn at the University of Minnesota. It was there he established his research interests to defining the role of renal sensory nerves in the development and maintenance of salt-sensitive hypertension and renal inflammation.

Hyndman received her PhD in 2008 in zoology from the University of Florida under the mentorship of David Evans, PhD. Her doctoral work focused on elucidating the evolution and physiology of the endothelin system in fishes. She completed postdoctoral research at the Medical College of Georgia under the mentorship of Jennifer Pollock, PhD. There, she tested the hypothesis that endothelin and nitric oxide work in the kidney to promote salt excretion. Now, as a principle investigator, she has two focused areas of research: Determining the physiological significance of the post-translational modification called lysine acetylation in the kidney; and determining if histone deacetylase enzymes are dysfunction in acute kidney injury leading to chronic kidney disease. Episodes of acute loss of kidney function are associated with increased risk of long-term decline in kidney function. Hyndman’s physiological studies are funded by a K01 and R03, while her acute kidney injury/chronic kidney disease studies are funded by the UAB-UCSD O’Brien Center Pilot and Feasibility Grant Award and thePriya Nagar Pilot Award for Kidney Research. She has published over 40 manuscripts, and serves on a number of national societies including the Media and Communications Committee of the American Society of Nephrology and the APS Career Opportunities in Physiology Committee. She currently serves on the VA Merit Nephrology Study Section and was recently appointed to the editorial board for the prestigious journal, Hypertension.